in lnfants of diabetic mothe following are true EXCEPT

(B). The probable pathogenic sequence is that maternal hyperglycemia causes fetal hyperglycemia, and the fetal pancreatic response leads to fetal hyperinsulinemia; fetal  hyperinsulinemia and hyperglycemia then cause Increased hepatic glucose uptake and glycogen synthesis, accelerated Ilpogenesls, and augmented protein synthesis. Related pathologic findings are hypertrophy and hyperplasia of the pancreatic islet [3 cells, increased weight of the placenta and Infant organs except for the brain, myocardial hypertrophy, increased amount of cytoplasm In liver cells, and extramedullary hematopoiesis. The incidence of congenital anomaliesis Increased 3-fold in infants of diabetic mothers; cardiac malformations (ventricular or atrial septal defect, transposition of the great vessels, truncus arteriosus, doubieoutlet right ventricle, tricuspid atresia, coarctation of the aorta) and iumbosacral agenesis are most common